Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN

Interleukin (IL)-18 facilitates both Th1 response and Th2 response.

Antioxidants, Free Full-Text

Contribution of mito-inflammation in neuronal disorders.

Frontiers Metabolic Reprogramming and Reactive Oxygen Species in T Cell Immunity

Mitochondrial control of inflammation

Cell Proliferation, Cell Biology Journal

Frontiers Mitochondrial DNA Release Contributes to Intestinal Ischemia/Reperfusion Injury

Elevated type I interferon responses potentiate metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling

Mitochondrial dysfunction in fibrotic diseases

Interferon Gamma Induces Reversible Metabolic Reprogramming of M1 Macrophages to Sustain Cell Viability and Pro-Inflammatory Activity - eBioMedicine

Free Fatty Acids Induce Apoptosis of Mammary Epithelial Cells of Ketotic Dairy Cows via the Mito-ROS/NLRP3 Signaling Pathway

Mitochondrial regulation of NLRP3 inflammasome activation and

IL-18 (Interleukin-18) Produced by Renal Tubular Epithelial Cells Promotes Renal Inflammation and Injury During Deoxycorticosterone/Salt-Induced Hypertension in Mice